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Showing posts with label Surgery. Show all posts
Showing posts with label Surgery. Show all posts

Thursday, April 5, 2018

12 Steps of Cesarean section




Preoperative preparation
·         Investigations: Blood : Hb, Coagulation profile, Serology, Blood group, Renal Fucntion test
·         Arrange II pint of whole blood and Cross match
·         Take Consent
·         Inform Anaesthesiologist
·         Inform Neonatologist/ Pediatrician

Intra operative preparations
·         Position: Supine
·         Wide bore canulation
·         Catherization
·         Preoperative medications including Antibiotics, gastroprotective agents
·         FHS Monitoring
·         Anaesthesia: Spinal/ General

Surgical Steps
Painting and draping  Lower abdominal inscision Soft tissue dissection and abdominal cavity reached. Doyen’s retracter is introduced and abdominal cavity is packed.  Identification of lower uterine segment and extraperitoneal incision is made over the uterine muscle. Membarane is ruptured, liquor suctioned and muscles spilt across. Fetal head  identified, doyen’s retracter removed and  baby  delivered. Doyen’s retractor reintroduced. Controlled cord traction of placenta.  Wound margin picked using hemostatic clamp  Uterine incision is sutured in 3 layers continuously using absorbable suture. Skin is closed in layers and dressing done. Wound packed.
12 steps of Cesarean section
1.       Painting and Draping
2.       Incision : Lower abdominal (Pfennestial) / midline vertical/ paramedian vertical
3.       Soft tissue dissection and opening of peritoneal cavity
4.       Doyen’s retracter is introduced and abdominal cavity is packed.
5.       Identification of Lower uterine segment and extraperitoneal incision is made over the uterine muscle.
6.       Membarane is ruptured, liquor suctioned and muscles spilt across.
7.       Fetal head is identified, Doyen’s retracter removed and  baby is delivered. Cords clamped and cut. Baby transferred.
8.       Doyen’s retractor reintroduced.
9.       Controlled cord traction performed and placenta removed. IV oxytoics provided.
10.   Wound margin picked using four Allis Forceps or green Armytage hemostatic clamp
11.   Uterine incision is sutured in 3 layers continuously using absorbable suture.
12.   Skin is closed in layers and dressing done. Wound packed.

Post operative care
·         Monitor: BP, Pulse, RR, Temperature, Fluid Input, Urine Output, Abdominal distension
·         Provide IV fluid, analgesics, Antibiotics, Oxytoxics
·         Breastfeed as early as mother is stable.
·         Ambulate as early as mother can tolerate
·         Start on sips as bowel sound appears, increase gradually to soft to solid food over 2-3 days.
·         Avoid coughing, straining and exertional work.
·         Wound dressing every 3-4 days and suture removal on Day 5-7.
·         Patient can be discharged once patient is stable and can take care of wound and diet.

Thursday, November 2, 2017

List of All named Hernias (Hernia Eponyms/Special Hernia)


Hernia is the abnormal protusion of a viscous or a part of it through an opening with a sac covering it. It could either be internal or external and not necessarily from the abdominal cavity. The most commonly encountered are the inguinal hernias and femoral hernia. But there are certain hernias that are named and arise from its specific location or contain specific content.

Hernia Eponyms on the basis of their similarity with the adjacent ones

Name of Hernia
Description
Bochdalek’s Hernia
Congenital Posterolateral Hernia of Diaphargm
Morgagni Hernia4
Through Larry’s space in Diaphargm
Larry’s Hernia4
Gibbon Hernia
Hernia with Hydrocele
Bergers Hernia
Hernia in Pouch of Douglas
Romberg Hernia1
Concurrent Ipsilateral Direct and indirect Inguinal Hernia
Saddle Hernia1
Dual Hernia1
Lumbar Hernia
Hernia in lumbar triangles of type Petit’s and Grynfelt
Obturator Hernia
Hernia through Obturator Canal
Grynfelt Hernia
Upper Lumbar triangle Hernia
Petit’s Hernia
Lower Lumbar Triangle Hernia
Femoral Hernia        
Hernia medial to Femoral Vein in Femoral canal
Cooper’s Hernia6
Femoral Hernia with two sacs
Bilocular femoral Hernia6
Cloquet’s Hernia
Hernia Through Pectineal aponeurosis
Callison-Cloquet Hernia
Through Pectineus Muscle and its fascia
Narath’s Hernia
Behind Femoral Artery in congenital dislocation of Hip
Hesselbach’s Hernia
Lateral to Femoral Artery
Serofini’s Hernia
Behind Femoral Vessels
Laugier’s Hernia
Through Lacunar Ligament
Teale’s Hernia5
In front of Femoral Vessels
Velpeau Hernia5
Richter’s Hernia
Part of circumference of bowel wall is entrapped
Littre’s Hernia
Contains Meckel Diverticulum
Sliding Hernia
Hernia with a part of the wall formed by the viscus
Hernia En Glissae
Hernia with a part of the wall formed by the viscus
Maydl’s Hernia2
Two loops of bowel in Hernia sac with remaining part in abdomen
W Hernia/Hernia in W2
Retrograde Hernia2
Two loops of bowel in Hernia sac with remaining part in abdomen
Phantom Hernia
Localised Muscle Bulge following Muscle Paralysis
Mery’s Hernia7
Through the perineal floor  
Perineal Hernia7
Spigelian Hernia
Hernia through Speligian Fascia
Sciatic hernia
Hernia through greater or lesser sciatic foramen
Little’s hernia3
Appendix in inguinal  Hernial Sac
Amyand hernia3
De Garengeots Hernia
Incarceration of appendix within Femoral Hernia
Beclards Hernia
Femoral Hernia Through Saphenous Opening
Barth’s Hernia
Hernia Between Adbominal wall and Persistent Vitellointestinal Duct
Holthouse’s Hernia                   
Inguinal Hernia That has turned outward into the groin
Grubers Hernia
Internal Mesogastric Hernia
Mesocolic hernia
Transmesenteric Hernia
Kronlem Hernia
Inguinoproperitoneal Hernia
Trietz Hernia
Paraduodenal Hernia
Rieux Hernia
Retrocaecal Hernia



Hernia Eponyms in alphabetical order

Name of Hernia
Description
Amyand hernia3
Appendix in inguinal hernia Sac
Barth’s Hernia
Hernia Between Adbominal wall and Persistent Vitellointestinal Duct
Beclards Hernia
Femoral Hernia Through Saphenous Opening
Bergers Hernia
Hernia in Pouch of Douglas
Bilocular femoral Hernia6
Femoral Hernia with two sacs
Bochdalek’s Hernia
Congenital Posterolateral Hernia of Diaphargm
Callison-Cloquet Hernia
Through Pectineus Muscle and its fascia
Cloquet’s Hernia
Hernia Through Pectineal aponeurosis
Cooper’s Hernia6
Femoral Hernia with two sacs
De Garengeots Hernia
Incarceration of appendix within Femoral Hernia
Dual Hernia1
Concurrent Ipsilateral Direct and indirect Inguinal Hernia
Femoral Hernia        
Hernia medial to Femoral Vein in Femoral canal
Gibbon Hernia
Hernia with Hydrocele
Grubers Hernia
Internal Mesogastric Hernia
Grynfelt Hernia
Upper Lumbar triangle Hernia
Hernia En Glissae
Hernia with a part of the wall formed by the viscus
Hesselbach’s Hernia
Lateral to Femoral Artery
Holthouse’s Hernia                   
Inguinal Hernia That has turned outward into the groin
Kronlem Hernia
Inguinoproperitoneal Hernia
Larry’s Hernia4
Through Larry’s space in Diaphargm
Laugier’s Hernia
Through Lacunar Ligament
Little’s hernia3
Appendix in inguinal  Hernial Sac
Littre’s Hernia
Contains Meckel Diverticulum
Lumbar Hernia
Hernia in lumbar triangles of type Petit’s and Grynfelt
Maydl’s Hernia2
Two loops of bowel in Hernia sac with remaining part in abdomen
Mery’s Hernia7
Through the perineal floor
Mesocolic hernia
Transmesenteric Hernia
Morgagni Hernia4
Through Larry’s space in Diaphargm
Narath’s Hernia
Behind Femoral Artery in congenital dislocation of Hip
Obturator Hernia
Hernia through Obturator Canal
Perineal Hernia7
Through the perineal floor
Petit’s Hernia
Lower Lumbar Triangle Hernia
Phantom Hernia
Localised Muscle Bulge following Muscle Paralysis
Retrograde Hernia2
Two loops of bowel in Hernia sac with remaining part in abdomen
Richter’s Hernia
Part of circumference of bowel wall is entrapped
Rieux Hernia
Retrocaecal Hernia
Romberg Hernia1
Concurrent Ipsilateral Direct and indirect Inguinal Hernia
Saddle Hernia1
Concurrent Ipsilateral Direct and indirect Inguinal Hernia
Sciatic hernia
Hernia through greater or lesser sciatic foramen
Serofini’s Hernia
Behind Femoral Vessels
Sliding Hernia
Hernia with a part of the wall formed by the viscus
Spigelian Hernia
Hernia through Speligian Fascia
Teale’s Hernia5
In front of Femoral Vessels
Trietz Hernia
Paraduodenal Hernia
Velpeau Hernia5
In front of Femoral Vessels
W Hernia/Hernia in W2
Two loops of bowel in Hernia sac with remaining part in abdomen


The number in superscript suggest that the hernias with the same number are actually same with various names.


Sources
SRB’s Manual Of Surgery
https://medicallegends.blogspot.com/2015/08/named-hernias.html
http://lessons4medicos.blogspot.com/2009/07/different-eponymous-types-of-hernia.html
Internet

Monday, September 4, 2017

Causes of Splenomegaly: Etiology and pathology




Enlargement of the size of the spleen beyond its normal shape and size and position is defined as splenomegaly. The normal anatomy of spleen is defined as a structure of 1inch x 3 inch x 5 inch, with weight of 7 ounces (~150-200g) in size and spans across 9-11 ribs in the left upper quadrant of the abdominal cavity. This is the rule of odd number in spleen (1, 3, 5, 7, 9, and 11).

Spleenomegaly   Infective    Bacterial     Enteric / paratyphoid    Typhus    Tuberculosis    Splenic Abscess    Septicemia   Viral     HIV    EBV (Infectious Mononucleosis)   Protozoal     Malaria    Schistosomiasis   Parasitic     Kalazar     Hydatid cyst   Spirochaetal     Syphilis    Weils disease  Hematological    RBC    Pernicious Anaemia     Polycythemia vera     Hereditary Spherocytosis    Autoimmune hemoltyic anaemia     Thalassemia    Sickle Cell Disease    Erythroblastosis Fetalis   WBC    CML    ALL    AML    CLL   Platelets    ITP  Metabolic    Rickets   Gauchers   Porphyria   Amyloidosis  Circulatory    Infarct   Portal hypertension   Segmental Portal Hypertension      Pancreatic Carcinoma     Splenic vein thrombosis  Collagen Vascular diseases   Still's disease   Felty Syndrome  Non Paracytic Cyst   Congenital   Acquired  Neoplastic    Angioma    Primary fibrosacroma   Hodkins Lymphoma    Myelofibrosis   Other lymphomas
Etiological Causes of Splenomegaly


The size of the spleen has to increase by at least 500g to called splenomegaly and has to be more than 1000g to define as massive splenomegaly. It is not necessary that every enlarged spleen will be palpable by clinical examination. The size of the spleen has to increase by 3-5 folds to be appreciated clinically. In terms of size it has to be greater than 10-11 cm in its greatest diameter to be defined as moderate splenomegaly and more than 20 cm in its largest dimension to be called as massive splenomegaly.

The cause of splenomegaly can be classified as either etiological causes or pathological cause.
Dividing the causes of splenomegaly etiologically, it could either be infective, hematological, metabolic, collagen vascular disease or other various causes. The most important one are obviously the infective and the hematological causes.

Infective   Bacterial    Enteric / paratyphoid   Typhus   Tuberculosis   Splenic Abscess   Septicemia  Viral    HIV   EBV (Infectious Mononucleosis)  Protozoal    Malaria   Schistosomiasis  Parasitic    Kalazar    Hydatid cyst  Spirochaetal    Syphilis   Weils disease
Infective causes of splenomegaly

Bacteria such as salmonella typhi/paratyphi, scrub typhus, mycobacterium tuberculosis, pneumococcal splenic abscess and septicemia by any organism can cause splenomegaly. HIV and EBV (infectious mononucleosis) are the viral causes of splenomegaly. Malaria and Schistosomiasis are few tropical protozoal disease to cause splenomegaly. Parasites like Leshmania donovani (kalazar) and echinococcus (hydatid cyst) are other causes. Syphilis and Weils (Leptospirosis) disease are some spirochetal conditions associated with splenomegaly.



Hematological Causes  RBC   Pernicious Anaemia    Polycythemia vera    Hereditary Spherocytosis   Autoimmune hemoltyic anaemia    Thalassemia   Sickle Cell Disease   Erythroblastosis Fetalis  WBC   CML   ALL   AML   CLL  Platelets   ITP
Hematological causes of splenomegaly

Hematological causes may be related to RBC, WBC or platelet. The condition where there is massive RBC destruction or massive extra medullary hemopoeisis is associated with splenomegaly. Most of the hemolytic causes of anemia such as hereditary spherocytosis, autoimmune hemolytic anemia, thalassemia, sickle cell disease, erythroblastosis fetalis have splenomegaly. Similarly faulty RBC synthesis in pernicious anemia leads to increased load in spleen to cause hemolysis. The polycythemia is associated with increased extra hemopoeitic synthesis of RBC. The Leukemia including  CML, ALL, AML, CLL will have splenomegaly. ITP also has splenomegaly.

Other causes  Metabolic    Rickets   Gauchers   Porphyria   Amyloidosis  Circulatory    Infarct   Portal hypertension   Segmental Portal Hypertension      Pancreatic Carcinoma     Splenic vein thrombosis  Collagen Vascular diseases   Still's disease   Felty Syndrome  Non Paracytic Cyst   Congenital   Acquired  Neoplastic    Angioma    Primary fibrosacroma   Hodkins Lymphoma    Myelofibrosis   Other lymphomas
Splenomegaly Causes other than infection and blood related

Metabolic causes such as rickets, Gaucher’s, porphyria, amyloidosis present with enlarged spleen clinically.  Infarction of spleen, portal hypertension associated with pancreatic carcinoma and splenic vein thrombosis are some of the circulatory or vascular causes. Collagen vascular diseases as Still's disease and Felty syndrome are other causes.  Non paracytic cyst either congenital or acquired cause splenomegaly.
Angioma , primary fibrosacroma, Hogdkins lymphoma, myelofibrosis are other lymphomas are the neoplastic causes of the condition. 

Saturday, September 2, 2017

History Writing: A case of Cellulitis

Italic words and parenthesized words and sentences are for readers purpose only not to be read when presenting a case. The letters In Blue are points of special interest to be discussed later.
All the patients may not have the same symptoms at presentation and the same risk factors, so history taking should always be INDIVIDUALISED than generalized to a standard sets of check list. It is always RECOMMENDED to ask the patient their problems and the question associated with the problem as per the need.
Interpersonal variations are always exists in the way the history is taken and written. Pattern, format and style of history taking and presenting are subject to change as per institutional protocol and region. Please kindly follow the system that is acceptable in your context.)

A case of bilateral limb swelling: Cellulitis

Name: Saraswoti Shrestha
Age: 44
Sex: Female
Religion: Hindu
Occupation: Farmer
Marital Status: married for 15 years
Address: Bhaktapur

Date of Admission: 21st November 2014
Date of Examination: 23rd November 2014
Mode of admission: Surgical OPD

Mrs. Saraswoti Shrestha, 44 years lady from Bhaktapur presented to surgery OPD with complains of

Chief complaints
                Pain and swelling of right leg for 11 days and left leg for 7 days
                Fever for 3 days  

History of Present Illness:
According to the patient she was in her usual state of health 11 days back then she gradually developed localized pain over the right lower leg around 10 cm below the knee joint. The pain was throbbing type, non-radiating. It was associated with itching sensation.  It was followed by swelling over that area, which gradually progressed downwards towards the ankle over a period of 3 days which made her leg swollen, red and shiny with patchy regions in-between.  Similarly, 7 days ago she developed similar painful area in the left lower limb inner aspect and gradually the limb swelled. The patient does not gives history of any trauma, insect bite or walking bare foot. (Risk factor of Cellulitis) No history of rashes, ulcer, vesicles, pus or discharge from the swollen area. No change in the color of overlying skin. (r/o Gangrene) She is unable to weight bear due to pain and swelling and has restricted her daily activity.

The swelling of limbs is associated with rise in body temperature for the last 3 days which was continuous and associated with chills. No rigor, no sweating and no rashes in other parts of the body.  However, the temperature is not documented. The patient does not give history of travel to other part of the country. (r/o Filaria)

Patient does not give history of prolonged immobility or any debilitating disease. (Risk factors for DVT) No history of severe pain even after walking a small distance. (Claudication)

The patient does not give history of chest pain, Shortness of breath, palpitation or hemoptysis. (r/o CHF)

No history of generalized weakness, lethargy, weight loss. No history of numbness or tingling sensation of the limbs. No known history of decreased sensation over the hand and feet. (Peripheral Neuropathy)

No history of burning micturition, urgency or frequency or excessive frothiness of urine. (r/o Nephrotic Syndrome)

No history of nausea, vomiting or pain abdomen. No history of altered bowel habit. No yellowish discoloration of skin or eyes. (r/o Liver failure)

History of past illness

The patient gives history of fever with sore throat two weeks back for which she took medication from the local medical shop which gradually subsided overtime.

She is known case of diabetes mellitus diagnoses 5 years back and is under oral medication. She does not give history of other chronic illnesses like HTN, Epilepsy or Tuberculosis. No history of any surgical intervention.

Personal history

Patient does not consume alcohol and is a nonsmoker.
She is non vegetarian and has normal bowel and bladder habit.
She has normal sleep pattern
She has a regular menstrual cycle occurring every 30 days, with menstrual bleeding for 2-3 days. She changes 2-3 partially soaked pads every day. She is not using Oral contraceptive Pills. (r/o DVT)

Family history

He has 8 members in the family.
No similar illness in the family.
No chronic illness like DM, HTN, TB or any cancers in the family.

Socioeconomic history

She belongs to a well sustained middle class family with good provision of clean drinking 
water and toilet facility.

Drug and allergy history

She has been taking medication for diabetes once daily in the morning. She does not consume other drug for any other chronic conditions.
No known history of allergy to any drug, food or any other substance.

Provisional Diagnosis
Bilateral Cellulitis with k/c/o Type II Diabetes Mellitus

Differential diagnoses
Erysipelas
Deep Vein Thrombosis
Filariasis
Nephrotic /Nephritic Syndrome
Congestive Heart Failure
Chronic Liver disease

*** Disclaimer: This is a hypothetical case and is not a real life scenario. However, the condition is so common and prevalent, it is a coincidence if it matches with the life of any. This case is solely for educational purpose with no intensions meant otherwise. ***